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Prolonged cognitive dysfunction in patient with splenial lesion of the corpus callosum caused by glufosinate ammonium poisoning
Hyun Jung Lee1, Jeong Ho Kang2
1 Department of Physical Medicine and Rehabilitation, Jeju National University Hospital, Republic of Korea, Korea
2 Department of Emergency Medicine, Jeju National University School of Medicine, Republic of Korea, Korea
Correspondence Address:
Jeong Ho Kang,
Department of Emergency Medicine, Jeju National University School of Medicine, 15, Aran 13-Gil, Jeju-si, Jeju-do, 63241
Korea
 Source of Support: None, Conflict of Interest: None DOI: 10.4103/2452-2473.309136
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Glufosinate ammonium (GLA) is widely used as a commercial herbicide in many countries. Neurotoxicity of GLA has been associated with serious neurological complications such as loss of consciousness, convulsions, and memory impairment. Late-onset memory impairment due to GLA-induced hippocampal lesions is the most distinct clinical feature in GLA poisoning. However, the lesion of the splenium of the corpus callosum (SCC) is a rare condition in GLA poisoning, so the clinical features are not well known. We report the case of a 57-year-old male patient who developed SCC damage after GLA poisoning. The patient had various late-onset neurotoxic symptoms, including prolonged overall cognitive dysfunction and psychosis-like symptoms. Emergency physicians should be aware that GLA-induced SCC lesions may be associated with various late-onset neurotoxic symptoms.
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